"In this study, using a mouse model for human breast cancer, we showed that TNBC's characteristics -- rapid tumor growth and metastasis -- is heavily dependent upon and fueled by markedly evaluated transcriptional activation of pro-inflammatory cytokines and chemokines," said Dr. Zhou. "Using a small molecule to target that gene transcriptional machinery, we further uncovered and validated a drug target for potential new treatment of TNBC, which account for more than 170,000 new breast cancers annually and which disproportionately affects women of African and Hispanic descent. While much more research needs to be done, our findings are very promising and give us hope in the overall war against breast cancer."
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http://www.mountsinai.org/about-us/newsroom/press-releases/discovery-of-root-cause-of-triple-negative-breast-cancer-may-lead-to-new-therapies
